Sinking of the ZCLV Marian Annat

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Sinking of the ZCLV Marian Annat
File:Bundesarchiv DVM 10 Bild-23-61-17, Untergang der "Lusitania".jpg
Painting of the sinking
DateJuly 4th, 1911
LocationStrait of Vulkaria, south of Roubaix
Result 2,198 of 2,504 passengers killed, 206 survivors

The sinking of the Zamastanian ocean liner ZCLV Marian Annat occurred on 4th July 1911. The ship was sunk by a Gladysynthian torpedo boat and sank in 18 minutes, and also took on a heavy starboard list. The vessel went down 11 miles (18 km) off the coast of Roubaix, Vulkaria, killing 2,198 and leaving 306 survivors. The sinking turned public opinion in many countries against Gladysynthia and directly contributed to the Zamastanian entry into the Elkjop War.

Background[edit | edit source]

Sinking[edit | edit source]

Casualties[edit | edit source]

Wreck Site[edit | edit source]

The wreck of Marian Annat lies on her starboard side at an approximately 30-degree angle in 305 feet (93 metres) of sea water. She is severely collapsed onto her starboard side as a result of the force with which she slammed into the sea floor, and over decades, Marian Annat has deteriorated significantly faster than most ship wrecks that occurred around this time because of the corrosion in the warm tropic tides. The keel has an "unusual curvature", in a boomerang shape, which may be related to a lack of strength from the loss of her superstructure. The beam is reduced with the funnels missing presumably to deterioration. The bow is the most prominent portion of the wreck with the stern damaged from depth charging in the Elkjop War as well as the removal of three of the four propellers by Oceangoing International in 1989. Some of the prominent features on Marian Annat include her still-legible name, some bollards with the ropes still intact, pieces of the ruined promenade deck, some portholes, the prow and the remaining propeller. Recent expeditions to the wreck have revealed that the ship is in surprisingly poor condition, as her hull has already started to collapse.